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We are working on further exploring our original discoveries in the fields of signal transduction and cell death control by linear ubiquitin chains and cellular iron metabolism. We also trying to make brand-new discoveries – i.e., independently of any previous research.

Access/Contact

Access/Contact
Kyoto University
Faculty of Medicine campus Bldg. C 5F
Yoshida-Konoe-cho, Sakyo-ku, Kyoto 606-8501,
JAPAN
TEL: +81-75-753-4673

Publications

HOIL-1L deficiency induces cell cycle alteration which causes immaturity of skeletal muscle and cardiomyocytes

Akagi K, Baba S, Fujita H, Fuseya Y, Yoshinaga D, Kubota H, Kume E, Fukumura F, Matsuda K, Tanaka T, Hirata T, Saito M, Iwai K, Takita J

scientific reports 2024 Apr 17;14(1):8871. DOI

Myofiber-type-dependent 'boulder' or 'multitudinous pebble' formations across distinct amylopectinoses

Mitra S, Chen B, Shelton JM, Nitschke S, Wu J, Covington L, Dear M, Lynn T, Verma M, Nitschke F, Fuseya Y, Iwai K, Evers BM, Minassian BA.

Acta Neuropathol. 2024 Feb 27;147(1):46. DOI

Attenuation of HOIL-1L ligase activity promotes systemic autoimmune disorders by augmenting linear ubiquitin signaling

Fuseya Y, Kadoba K, Liu X, Suetsugu H, Iwasaki T, Ohmura K, Sumida T, Kochi Y, Morinobu A, Terao C, Iwai K.

JCI Insight 2024 Feb 8;9(3):e171108 DOI

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